Signs of DIC can either be overt or covert and this is because it can be full brown hemorrhage or it can subtly develop. Laboratory tests say that the two basic causal factors are micro emboli that results from unchecked clotting process and also the unchecked dissolution of clot that leads to the activation of the system’s coagulation cascade. Sometimes DIC disease can be chronic something that means that it never comes to the surface but remains on a low level or the coagulation activation process runs intermittently. Chronic DIC is caused by solid malignant tumors, a state that enables blood to clot more (Geiter 2003).
It is good to differentiate the actual symptoms of DIC because some disorders resemble those of DIC. Its treatment consists of three basic modalities which should run concurrently. The first one is trigger removal, here the causal factor is identified and removed otherwise the treatment would be unproductive. The second treatment is perfusion maintenance. Supportive care provided should be designed in such a way that it boosts tissue perfusion. Again, oxygen saturation cardiac saturation, cardiac support and blood pressure should be controlled. The third and the last one is to balance restoration.
This involves balancing of the clot formation, dissolution and inhibition (Geiter, 2003) Disseminated intravascular coagulopathy as a disease that affects intravascular system is a bit complicated disease that requires proper diagnosis to reduce the chances of confusing other disorders with those of DIC. It is not exactly known the mechanism that triggers off DIC but its causal factors are known. Its treatment involves three stages namely trigger removal, perfusion maintenance and balance restoration.
Reference:
Geiter Henry. 2003. Disseminated intravascular coagulopathy. Clinical Dimension. Vol. 22, No: 3.